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These findings suggest that miRNAs may be involved in vitiligo pathogenesis via the modulation of vital genes expression in melanocytes and serve as novel therapeutic targets for vitiligo therapy

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By contrast, inflammatory activation and progressive oxidative injury appear to function predominantly as downstream processes that exacerbate mitochondrial dysfunction and cell damage after the initial loss of calcium homeostasis has occurred

In addition, we observed decreased esterfied PUFAs in all GP classes, glycerolipids (GL) as well as in lyso-species (Fig